The etiological origin and the possibility of pharmacological intervention of autism represent two areas of great scientific interest and on which research has not yet provided conclusive results. However, as already highlighted by CREA in many publications on this site, there are more and more frequent studies that place in genetics some important causal and therapeutic principles.
The evolution of neuroscientific knowledge seems to indicate a pathogenetic area common in intellectual disabilities, autism and schizophrenia. All genetic vulnerability and gene-environment interaction may play a decisive role in the formation of circuits for memory and other cognitive functions.